BIOSKINCARE – our powerful natural cream can be applied to mild actinic keratosis lesions on the skin (solar keratosis) causing them to heal, regress and inhibit their progresion to invasive skin cancer.
Our BIOSKINCARE cream is a biological solution for mild actinic keratosis
- Grinds down mild actinic keratosis through enzyme dissolution of keratin and abnormal tissues and prevents the growth of new actinic keratosis scaly formation.
- Acts like a non trauma exfoliation by eliminating dead cells and scaly lesions before they grow.
- Also destroys pathogens, heals and regenerates damaged tissue.
- It nourishes your skin and protects it from free radicals.
- Improves the skin’s elasticity, and leaves it silky soft and soothed.
BIOSKINCARE is a non prescription, natural skin care remedy, with absolutely no side effects.
What is Actinic Keratosis (AK)?
It is a scaly or crusty bump that arises on the skin surface. The base may be light or dark, tan, pink, red, or a combination of these, or the same color as your skin. The scale or crust is horny, dry, and rough, and is often recognized by touch rather than sight. Occasionally it itches or produces a pricking or tender sensation. It can also become inflamed and surrounded by redness. In rare instances, actinic keratosis can even bleed.
The skin abnormality or actinic keratosis lesion develops slowly and generally reaches a size from an eighth to a quarter of an inch. Early on, it may disappear only to reappear later. You will often see several actinic keratosis at a time.
An actinic keratosis is most likely to appear on the face, ears, scalp, neck, backs of the hands and forearms, shoulders, and lips – the parts of the body most often exposed to sunshine. The growths may be flat and pink or raised and rough.
Actinic keratosis are lesions of the skin induced by solar radiation, and are usually harmless but there is a small risk that they may progress to a form of invasive skin cancer called squamous cell carcinoma. It is by far the most common skin lesion with malignant potential.
Anyone that has actinic keratosis has received serious skin damage caused by nothing less than the harsh UV rays of the sun, which obviously makes the sufferer more susceptible to other sun related skin diseases and can ultimately end up in skin cancer: squamous cell carcinoma as well as basal cell carcinoma cancer. If treated early, almost all actinic keratosis can be eliminated without becoming skin cancers.
Who is Prone to Actinic Keratosis?
Actinic keratosis is seen in people with fair-skin color on skin areas that have been frequently exposed to solar radiation for a long period of time, generally since childhood (from sunbathing, sunbed use, outdoor work or recreational activities).
Australia is the country with the highest skin cancer rate in the world, and there the prevalence of actinic keratosis among adults older than 40 years has been reported to range from 40-60%.
AKs usually appear after age 40 or 50 because they take many years to develop. The Skin Cancer Foundation reports more than ten million Americans currently have actinic keratosis.
What Causes Actinic Keratosis?
Keratosis is the build up of keratin on the skin and can be due to many causes, however, the most common cause is over-exposure of skin areas to the sun. And these areas usually become sore and inflamed.
What are the symptoms of Actinic Keratosis?
Actinic keratosis primarily appears on the areas that are most frequently in direct contact with the suns UV rays. The main areas where it appears are on the face, ears, a bald scalp, neck, backs of the hands and arms and the lips. In some cases it has been known to appear on the buttocks and thighs.
- Lesions on the lips are known as Actinic cheilitis (it is also the most aggressive form) characterized by a white scaling and thickening of the lips in affected areas. On the head and neck the small lesions are usually flat, while on the arms and hands they are elevated. So if one stays away from the sun it is possible for the skin to heal itself gradually and get rid of the keratosis build up.
Individuals with light skin are most likely to develop actinic keratosis. The condition is directly associated to sun exposure, and therefore is sometimes called “solar keratosis”. In fact, fair-skinned individuals living in sunny climates have over 50 percent chance of developing actinic keratosis. Individuals aged 40 and up are at high risk, with more men affected by this common, pre-cancerous condition than women. Furthermore, regardless of age, all individuals with blue eyes and or childhood freckling are at a higher risk for actinic keratosis.
Progression of Actinic Keratosis Skin Lesions into Pre-Cancer Lesions
Usually, lesions develop as a single, small plaque on the face of people aged 20-30 years with light coloring and significant sun exposure; they gradually progress in sun-exposed areas, such as the nose, the forehead, and the cheeks.
During periods of depressed immunity, the visible and the subclinical lesions flare and become inflammatory red and scaly. This depression of the immune system can occur following intense ultraviolet light exposure or with systemic chemotherapy for other carcinomas.
Over the years, the lesions gradually progress, and approximately 1 in 20 lesions eventually turns into invasive carcinomas. These lesions are typically the more red and inflamed, elevated, and indurated lesions. In very hyperkeratotic lesions, the invasive component of the carcinoma is at the base of the lesion and is often not seen until a physician removes the overlying plaque.
The morphology of atypical cells in both actinic keratosis and squamous cell carcinoma is identical. The risk of progression to squamous cell carcinoma (the most common form of skin cancer) is minimal. But up to 60% of squamous cell carcinoma cases begin as actinic keratosis. Regression of actinic keratosis occurs when sun exposure is decreased and mild lesions are healed.
How to Treat Actinic Keratosis
Treatment is often indicated to prevent the cells from invading the dermis and possibly metastasizing. If a lesion exhibits evidence of possible dermal invasion, such as marked erythema (redness of the skin caused by dilatation and congestion of the capillaries, often a sign of inflammation or infection) ulceration, tenderness, bleeding, and especially hardening of a normally soft tissue, a physician will consider performing a biopsy.
Medical care for Actinic Keratosis Lesions
As lesions progress, they can be treated with topical 5-fluorouracil. This medication is applied twice daily for a period of 3-4 weeks for facial lesions. Other body sites require longer treatment, with the arms needing 6-8 weeks. Treating the entire area of involvement is best.
During the treatment phase, the actinic keratosis lesions become increasingly red, and small subclinical lesions become visible. This treatment can be temporarily disfiguring, with inflammatory and red ulcerations and crust formation. However, if the patient completes the treatment, the lesions usually heal within 2 weeks of stopping treatment, the complexion is smooth, and the actinic keratosis are improved.
Topical 5-fluorouracil treatment can be repeated on several occasions. The few residual lesions that are not completely cleared with 5-fluorouracil can then be treated surgically.
Photodynamic therapy (PDT) can also be used to treat multiple actinic keratoses. In this treatment, topical 5-aminolevulinic acid accumulates preferentially in the dysplastic cells. On exposure to irradiation with light of an appropriate wavelength, oxygen free radicals are generated and cell death results. Patients experience pain in the areas treated, which is similar in quantity to the pain resulting from topical 5-fluorouracil. One treatment with PDT appears to be as effective as 5-fluorouracil and may be considered in patients with contact sensitivity to 5-fluorouracil.
Freezing with liquid nitrogen (Cryotherapy). This is an effective treatment which does not normally leave a scar, but it can be painful. (see Patient Information Leaflet on Cryotherapy)
Surgical removal. This requires local anaesthetic, after which the actinic keratosis can be scraped off with a sharp spoon-like instrument (a curette), or it can be cut out and the wound closed with stitches. Surgical removal leaves a scar but provides a specimen that can be analysed in the laboratory to confirm the diagnosis.
Surgical Care of Actinic Keratosis
- Early lesions of actinic keratoses can be treated with destruction. Cryotherapy with liquid nitrogen is the most effective and practical method for treating actinic keratoses when a limited number of lesions are present. Liquid nitrogen, applied by swab or spray, is used for several seconds. As the cryotherapy wound heals, the actinic keratosis is sloughed off. These patients may need periodic re-treatment for small recurrences or for new lesions. Often, semiannual or annual monitoring and treatment are warranted.
- Physical destruction using electrodesiccation (destruction of lesions or sealing off of blood vessels by monopolar high-frequency electric current) and curettage (scrapping of tissues with an instrument shaped like a scoop or spoon) is particularly effective when a person has hyperkeratotic lesions (scaly or hardenned keratin proteins on the surface of the skin).
- Chemical peels may be used in patients with a number of facial lesions.
- Dermabrasion may be used.
Adverse reactions such as scarring, textural changes, infection, and pigmentation alteration rarely occur.
If a lesion does not respond to treatment, obtaining a biopsy of the lesion should be considered to be certain that the lesion is not an invasive squamous cell carcinoma.
Cryosurgery may result in wounds, require time for wound healing, and may yield hypopigmentation (white spots) or scarring and although those side effects were acceptable in the past, many people now developing actinic keratosis have little tolerance for any time for wound healing or any cosmetic changes. The best therapy is thus one that addresses minor actinic keratosis scaly formations early, and here is where BIOSKINCARE plays a significant role.
Try BIOSKINCARE CREAM
And Get Your Skin Healthy Again Quickly!
BIOSKINCARE has a two fold effect: (1) Purifies the cells & decongests tissues as it helps to eliminate waste, toxins, actinic keratosis crusts and all damaged structures. (2) Favors cellular regeneration.
It leaves your skin smooth, refreshed, soft and with use over a period of time it takes away all imperfections and blemishes: acne spots, scars, keratosis bumps, razor nicks and burns, actinic keratosis lesions, roughness, dryness, eczema, dermatitis, the effects on the skin of radiotherapy for cancer, blisters, scrapes, cuts, and the list can go on and on… It also brightens and enhances your complexion.
BIOSKINCARE is a non prescription, natural skin care remedy, with absolutely no side effects.
The cream is made with a natural biological ingredient, collected from live land snails, the same they use to quickly repair their own skin and shell when damaged. It is contained in the mucin secreted by the snails, (we gather it through a process that is safe for them). It is a viscose liquid that is made into an odorless white cream with no alien, synthetic chemicals. Analysis of the mucin has determined that it contains activators of skin growth factors and antimicrobial peptides.
Also presents natural anti-inflammatory and immune modulating proteins, glycoproteins, glycosaminoglycans (a group of polysaccharides with high molecular weight that contain amino sugars and often form complexes with proteins. Also called mucopolysaccharide) and copper.
The results are:
With the use of BIOSKINCARE for a month you may remain free of Actinic Keratosis for months and possibly years. Or you may need to repeat the treatment every 6 months. However, individuals with significant photodamage and those who fail to minimize sun exposure are at increased risk for the development of additional Actinic Keratosis, as well as skin cancer.
Preventing Actinic Keratosis
- Apply a broad spectrum sunscreen (protects against both UVA and UVB rays) that has a Sun Protection Factor (SPF) of 15 or more, every day.
- Avoid deliberate tanning. Even when wearing sunscreen, sunbathing is not safe. Tanning beds and sunlamps are not safe alternatives because they too emit UV rays.
- Stay out of the sun between 10 a.m. and 4 p.m. This is when the sun’s rays are the strongest. Seek shade whenever possible. If you don’t see your shadow or your shadow is shorter than you, it is important to seek cover.
- Wear protecting clothing when you are on the sun.
- A wide-brimmed hat is a must to protect your face and neck from the harmful rays. Wear sunglasses that provide 100% UV protection.
BIOSKINCARE will not heal severe keratosis that may have already a cancerous formation.
Grinds down mild actinic keratosis scales and horny crusts through enzyme dissolution of keratin and hydrolysis of abnormal tissues, and prevents the growth of new actinic keratosis scaly formations.
About Actinic Keratosis
Web Links of Interest:
- Clears Actinic Keratosis Crusts
- Prevents Actinic Keratosis
- Makes AK Lesions Regress
- Powered by BIOCUTIS® Serum
- Supports Your Immune System
- Heals Skin Quickly
- Repairs Sun Damaged Skin
- The Most Powerful Antioxidants
- Moisturizes Deep Within The Skin
- Removes Skin Blemishes
- No Side Effects
- Money Back Guarantee
Ingredients in BIOSKINCARE
- Distilled Water (solvent)
- Peg-4 Olivate (emulsifier derived from Olive Oil)
- Sorbitan Olivate from Olive Oil (moisturizer)
- Squalane from Olive Oil (natural moisturizer and antioxidant)
- Hyaluronic Acid (in solution – moisturizer)
- Biocutis® Serum: Helix Aspersa Müller Glycoconjugates
- Glycerin (humectant)
- SUPRAPEIN™, a synergistic combination of botanical extracts that yields a natural preservative with a broad-spectrum of antimicrobial activity.
“I have been clearing up a number of Actinic Keratosis with Folic Acid, B12, B17 and Aloe vera gel over almost 10 years. The BIOSKINCARE Cream appears as though it will clear the remains up within 2-3 weeks. A significant lesion appeared last May as a result of the Aloe Vera etc treatment and although it has gradually been diminishing, there has been far greater change in 1 week with BIOSKINCARE than the last 9 months.”
Peter Melvelle. NSW, Australia
“Several years of going to dermatologists who used corrosive treatments on me for extensive actinic keratosis have not even remotely given me the results I’ve experienced from using BIOSKINCARE for just 3 weeks so far. I look forward to seeing the results I’ll get after using BIOSKINCARE several more weeks!”
Suzanne H. Kahn. GA, US
“I have been using your product for actinic keratosis for over a month and have been very pleased with it’s healing qualities. I’m excited to see how much more my skin can improve with it’s use.”
Deborah Leonard. London
“I used this on my sunburn, and it was very effective in healing it quickly!”
Erin Liedel. Boston, MA, USA
Progression of Actinic Keratosis to Skin Cancer
In 2009, Criscione and colleagues conducted a study examining the progression of actinic keratoses to squamous cell carcinoma and basal cell carcinoma. The study examined 7784 actinic keratoses in a high-risk population and found that nearly 65% of primary squamous cell carcinomas and 36% of primary basal cell carcinomas arise from clinically diagnosed actinic keratoses. Furthermore, risk of progression of actinic keratosis to squamous cell carcinoma was 0.60% at 1 year and 2.57% at 4 years, which is 6-8 times more frequent than has been reported in some prior studies. The risk of progression of actinic keratosis to basal cell carcinoma was 0.48% at 1 year and 1.97 at 4 years, although it is acknowledged that it is possible that many of these lesions may have been previously mistaken for actinic keratoses.
Criscione VD, Weinstock MA, Naylor MF, Luque C, Eide MJ, Bingham SF. Actinic keratoses: Natural history and risk of malignant transformation in the Veterans Affairs Topical Tretinoin Chemoprevention Trial. Cancer. Jun 1 2009;115(11):2523-30.
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